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|Appears in Collections:||Aquaculture Journal Articles|
|Peer Review Status: ||Refereed|
|Title: ||Novel non-motile phenotypes of Yersinia ruckeri suggest expansion of the current clonal complex theory|
|Author(s): ||Tinsley, John W|
Austin, Dawn A
Lyndon, Alastair R
|Contact Email: ||firstname.lastname@example.org|
|Keywords: ||Yersinia ruckeri|
|Issue Date: ||Apr-2011|
|Citation: ||Tinsley JW, Austin DA, Lyndon AR & Austin B (2011) Novel non-motile phenotypes of Yersinia ruckeri suggest expansion of the current clonal complex theory, Journal of Fish Diseases, 34 (4), pp. 311-317.|
|Abstract: ||The biochemical and cell surface characteristics of 63 non-motile isolates of Yersinia ruckeri from various sources were compared using the API 20E rapid identification system and conventional phenotypic methods. Eight individual phenotypic groups were observed from the data set. All the non-motile isolates were from rainbow trout whereas novel phenotypes were observed in other fish species. Non-motile isolates were not exclusively observed from serogroup O1; membership of biotype 2 was recorded for representatives from serogroups O2 to O7. Variations in phenotypes highlights that new clonal groups are arising and that the current typing scheme requires expansion. Previously, it was hypothesised that disease was caused by a few virulent clones, data in this paper suggests that this is not the case. The lipopolysaccharide (O antigen) type in the non-motile biotype was different to other isolates of Y. ruckeri.|
|Type: ||Journal Article|
|DOI Link: ||http://dx.doi.org/10.1111/j.1365-2761.2011.01237.x|
|Rights: ||The publisher does not allow this work to be made publicly available in this Repository. Please use the Request a Copy feature at the foot of the Repository record to request a copy directly from the author; you can only request a copy if you wish to use this work for your own research or private study.|
|Affiliation: ||Heriot-Watt University|
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