Please use this identifier to cite or link to this item:
http://hdl.handle.net/1893/28366
Appears in Collections: | Psychology Journal Articles |
Peer Review Status: | Refereed |
Title: | Cumulative culture and explicit metacognition: a review of theories, evidence and key predictions |
Author(s): | Dunstone, Juliet Caldwell, Christine A |
Issue Date: | 4-Dec-2018 |
Date Deposited: | 10-Dec-2018 |
Citation: | Dunstone J & Caldwell CA (2018) Cumulative culture and explicit metacognition: a review of theories, evidence and key predictions. Palgrave Communications, 4 (1), Art. No.: 145. https://doi.org/10.1057/s41599-018-0200-y |
Abstract: | A variety of different proposals have attempted to explain the apparent uniqueness of human cumulative culture as a consequence of underlying mechanisms that are also assumed to be uniquely well-developed in humans. Recently, Heyes and colleagues have proposed explicit (or Type 2) metacognition as a key feature of human cognition that might enable cumulative culture. In the current review we examine these arguments, and consider their plausibility. Firstly we consider whether distinctions between cognitive processes described as explicit/implicit, and Type 1/2 (or Systems 1/2), do indeed capture features that distinguish processes specific to human cognition, versus those that are shared with other species. In particular we consider whether this applies to distinctions relating to metacognitive processes. We also consider the ways in which explicit metacognitive processing might plausibly facilitate cumulative culture. We categorise the potential benefits as either optimising receiver behaviour, or optimising sender behaviour. Within both of these categories benefits could arise as a consequence of more effective representation of either one’s own knowledge state, or that of others. We evaluate the current state of evidence supporting each of these potential benefits. We conclude by proposing methodological approaches that could be used to directly test the theory, and also identify which (if any) of the possible causal mechanisms may be implicated. |
DOI Link: | 10.1057/s41599-018-0200-y |
Rights: | This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. |
Licence URL(s): | http://creativecommons.org/licenses/by/4.0/ |
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